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Olive
oil
polyphenols
protect
endothelial
dysfunction
induced
by
high
glucose
and
free
fatty
acids
Olive
oil
polyphenols
protect
endothelial
dysfunction
induced
by
high
glucose
and
free
fatty
acids
上传时间:2016-07-01
页数:2
格式:PDF
浏览: 1
Polyphenol fraction of extra virgin
olive
oil
protects against
endothelial
dysfunction
induced
Polyphenol fraction of extra virgin
olive
oil
protects against
endothelial
dysfunction
induced
上传时间:2016-01-13
页数:7
格式:PDF
浏览: 1
Aldolase
B
Knockdown Prevents
High
Glucose-Induced
Methylglyoxal Overproduction and Cellular
Aldolase
B
Knockdown Prevents
High
Glucose-Induced
Methylglyoxal Overproduction and Cellular
上传时间:2016-10-24
页数:10
格式:DOC
浏览: 0
Role of
free
fatty
acids
in
endothelial
dysfunction
Role of
free
fatty
acids
in
endothelial
dysfunction
上传时间:2018-06-05
页数:15
格式:PDF
浏览: 1
Daming capsule restores
endothelial
dysfunction
induced
by
high
-fat diet
Daming capsule restores
endothelial
dysfunction
induced
by
high
-fat diet
上传时间:2016-11-24
页数:8
格式:DOC
浏览: 0
The effects of metabolites of
polyphenols
and vitamin E on modulating the
high
glucose-induced
The effects of metabolites of
polyphenols
and vitamin E on modulating the
high
glucose-induced
上传时间:2023-03-21
页数:36
格式:PDF
浏览: 0
Modelling the influence of palmitic and oleic
acids
on
free
fatty
acids
Modelling the influence of palmitic and oleic
acids
on
free
fatty
acids
上传时间:2016-02-23
页数:27
格式:DOC
浏览: 2
Angiotensin II mediates the
high-glucose-induced
endothelial
-to-mesenchymal transition in human
Angiotensin II mediates the
high-glucose-induced
endothelial
-to-mesenchymal transition in human
上传时间:2016-11-01
页数:7
格式:DOC
浏览: 1
Mitochondrial
dysfunction
is
induced
by
high
levels of
glucose
and
free
fatty
acids
in 3T3-L1
Mitochondrial
dysfunction
is
induced
by
high
levels of
glucose
and
free
fatty
acids
in 3T3-L1
上传时间:2015-12-25
页数:9
格式:PDF
浏览: 2
Research of the action of the methylic esters of the
fatty
acids
from the extra-virgin
olive
oil
Research of the action of the methylic esters of the
fatty
acids
from the extra-virgin
olive
oil
上传时间:2014-12-29
页数:8
格式:DOC
浏览: 0
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